Ferroptosis, a new non-necrotizing programmed cell death (PCD), is driven by iron-dependent phospholipid peroxidation.Ferroptosis plays a key role in secondary traumatic brain injury and secondary cerini coffee and gifts products spinal cord injury and is closely related to inflammation, immunity, and chronic injuries.The inhibitors against ferroptosis effectively improve iron homeostasis, lipid metabolism, redox stabilization, neuronal remodeling, and functional recovery after trauma.
In this review, we elaborate on the latest molecular mechanisms of ferroptosis, emphasize its grand love red heart reposado tequila role in secondary central nervous trauma, and update the medicines used to suppress ferroptosis following injuries.